Biochemistry of Oxidative Stress

Group leader

Joaquim Ros Salvador

973 702 275

joaquim.ros(ELIMINAR)@udl.cat

Joaquim Ros Salvador

Publications (91)

Publication Search

Medina-Carbonero, M; Sanz-Alcázar, A; Britti, E; Delaspre, F; Cabiscol, E; Ros, J; Tamarit, J

Mice harboring the FXN I151F pathological point mutation present decreased frataxin levels, a Friedreich ataxia-like phenotype, and mitochondrial alterations.

Cellular And Molecular Life Sciences 79 74-74. .

[doi:10.1007/s00018-021-04100-5]

Tamarit J; Britti E; Delaspre F; Medina-Carbonero M; Sanz-Alcázar A; Cabiscol E; Ros J

Mitochondrial iron and calcium homeostasis in Friedreich ataxia.

IUBMB LIFE 73 543-553. .

[doi:10.1002/iub.2457]

Britti E; Delaspre F; Sanz A; Medina-Carbonero M; Llovera M; Purroy R; Mincheva-Tasheva S; Tamarit J; Ros J

Calcitriol increases frataxin levels and restores mitochondrial function in cell models of Friedreich Ataxia.

BIOCHEMICAL JOURNAL 478 1-20. .

[doi:10.1042/BCJ20200331]

Britti, E; Delaspre, F; Tamarit, J; Ros, J

Calpain-Inhibitors Protect Frataxin-Deficient Dorsal Root Ganglia Neurons from Loss of Mitochondrial Na + /Ca 2+ Exchanger, NCLX, and Apoptosis.

NEUROCHEMICAL RESEARCH 46 108-119. .

[doi:10.1007/s11064-020-03020-3]

Rodríguez-Pascau L; Britti E; Calap-Quintana P; Dong YN; Vergara C; Delaspre F; Medina M; Tamarit J; Pallardó FV; Gonzalez-Cabo P; Ros J; Lynch DR; Martinell M; Pizcueta P

PPAR gamma agonist leriglitazone improves frataxin-loss impairments in cellular and animal models of Friedreich Ataxia.

NEUROBIOLOGY OF DISEASE 148 105162-105162. .

[doi:10.1016/j.nbd.2020.105162]

Rodríguez Colman MJ; Ros J; Cabiscol E

Mitochondrial Localization of the Yeast Forkhead Factor Hcm1.

INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES 21 1-11. .

[doi:10.3390/ijms21249574]

Purroy, R; Medina-Carbonero, M; Ros, J; Tamarit, J

Frataxin-deficient cardiomyocytes present an altered thiol-redox state which targets actin and pyruvate dehydrogenase.

Redox Biology 32 101520-101520. .

[doi:10.1016/j.redox.2020.101520]

Britti, E; Ros, J; Esteras, N; Abramov, AY

Tau inhibits mitochondrial calcium efflux and makes neurons vulnerable to calcium-induced cell death.

CELL CALCIUM 86 102150-102150. .

[doi:10.1016/j.ceca.2019.102150]

Guerrero-Gomez, D; Mora-Lorca, JA; Saenz-Narciso, B; Naranjo-Galindo, FJ; Munoz-Lobato, F; Parrado-Fernandez, C; Goikolea, J; Cedazo-Minguez, A; Link, CD; Neri, C; Sequedo, MD; Vazquez-Manrique, RP; Fernandez-Suarez, E; Goder, V; Pane, R; Cabiscol, E; Askjaer, P; Cabello, J; Miranda-Vizuete, A

Loss of glutathione redox homeostasis impairs proteostasis by inhibiting autophagy-dependent protein degradation

CELL DEATH AND DIFFERENTIATION 26 1545-1565. .

[doi:10.1038/s41418-018-0270-9]

Vall-llaura, N; Mir, N; Garrido, L; Vived, C; Cabiscol, E

Redox control of yeast Sir2 activity is involved in acetic acid resistance and longevity

Redox Biology 24 101229-101229. .

[doi:10.1016/j.redox.2019.101229]

Elena Britti; Noemi Esteras Gallego; Joaquim Ros; Andrey Y. Abramov

Mitochondrial Calcium Deregulation in Tau K18-Treated Cortical Neurons and Astrocytes

BIOPHYSICAL JOURNAL 116 270-271. .

[doi:10.1016/j.bpj.2018.11.1464]

Britti, Elena; Delaspre, Fabien; Tamarit, Jordi; Ros, Joaquim

Mitochondrial calcium signalling and neurodegenerative diseases

Neuronal Signaling 2 20180061-20180061. .

[doi:10.1042/NS20180061]